By Suna F. Kiraç
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Additional resources for Advances in the Diagnosis of Coronary Atherosclerosis
184) (Fig. 3) (unpublished data). Again, these findings suggest that TRAIL may not be a good candidate as a biomarker of early atherosclerotic lesions. Fig. 3. The association of TRAIL with endothelial function in 109 healthy subjects. Flow-mediated dilatation (FMD) was used to evaluate endothelial function in 109 healthy subjects. 148). 28 Advances in the Diagnosis of Coronary Atherosclerosis 3. Conclusions Recent emerging evidence has suggested the definite involvement of TRAIL in cardiovascular diseases.
3). 5. References Abedin, M, et al. (2007). Relation of osteoprotegerin to coronary calcium and aortic plaque (from the Dallas Heart Study). Am J Cardiol, Vol. 99, No. 4, (February 2007) pp. 513518, ISSN 0002-9149 Afford, SC, Adams, DH. (2005). Following the TRAIL from hepatitis C virus and alcohol to fatty liver. Gut, Vol. 54, No. 11, (October 2005) pp. 1518-1520, ISSN 0017-5749 Alladina, SJ, et al. (2005). TRAIL-induced apoptosis in human vascular endothelium is regulated by phosphatidylinositol 3-kinase/Akt through the short form of cellular FLIP and Bcl-2.
Relationship of serum osteoprotegerin levels with coronary artery disease severity, left ventricular hypertrophy and C-reactive protein. Clin Sci (Lond), Vol. 108, No. 3, (December 2004) pp. 237-243, ISSN 0143-5221 Ross, R. (1993). The pathogenesis of atherosclerosis: a perspective for the 1990s. Nature, Vol. 362, No. 6423, (April 1993) pp. 801-809, ISSN 0028-0836 Sandoo, A, et al. (2010). The endothelium and its role in regulating vascular tone. Open Cardiovasc Med J, Vol. 4, No. (February 2011) pp.